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Arcese Quarter Horses USA Surpasses Two Million

Arcese Quarter Horses USA Surpasses Two Million

Quarter Horse News / by john.williams@morris.com (Press Release)

Eleuterio Arcese
The reining world is accustomed to seeing milestones shattered during the National Reining Horse Association (NRHA) Futurity & Adequan® North American Affiliate Championship Show (NAAC). This year’s event proved no different with an NRHA first going into the record books: Arcese Quarter Horses USA became NRHA’s first Two Million Dollar Owner. The operation, based in Weatherford, Texas, officially has $2,007,400 in earnings.

ARC Walla Dun Did It and NRHA Three Million Dollar Rider Andrea Fappani added more than $35,000 to Arcese’s total with a go-round placing and a top ten finish in the Level (L) 4 Open Finals. To make the accomplishment that much sweeter, the stallion was sired by their very own Walla Walla Whiz.

Eleuterio Arcese, of Arcese Quarter Horses USA, has worked to promote the sport of Reining in Europe for more than twenty years through his involvement with NRHA, the Italian Reining Horse Association, and the Italian Quarter Horse Association. The NRHA Hall of Fame member has been host to multiple reining clinics and sponsored major NRHA and AQHA reining events in Europe. As an avid breeder of performance horses, Arcese has influenced breeding programs at his facilities in Italy as well as in the United States.

Outstanding reining horses owned by Arcese Quarter Horses USA include 2007 NRHA Futurity, 2008 NRBC and 2008 NRHA Derby Champion Wimpys Little Chic, plus the likes of Americasnextgunmodel, Gunnatrashya, Walla Walla Whiz, Custom Mahogany, Electrical Flash, Whizs Chic A Dee, Yankee Gun, Saturdaynight Custom and Custom Lena. Eleuterio, along with his children Leonardo, Paola and Matteo, have created a reining legacy in Arcese Quarter Horses USA.

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Evaluation of the safety of a combination of oral administration of phenylbutazone and firocoxib in horses.

Evaluation of the safety of a combination of oral administration of phenylbutazone and firocoxib in horses.

J Vet Pharmacol Ther. 2013 Dec 20;

Authors: Kivett L, Taintor J, Wright J

Abstract
Simultaneous administration of a nonselective COX inhibitor and a COX-2 specific NSAID has not been previously reported in horses. The goal of this study was to determine the safety of a 10-day dosage regimen of phenylbutazone and firocoxib, both at their standard dosages, in horses. Six horses were administered 2.2 mg/kg of phenylbutazone and 0.1 mg/kg of firocoxib by mouth, daily for 10 days. Horses were assessed daily for changes in behavior, appetite, fecal consistency, signs of abdominal pain, and oral mucous membrane ulceration. Horses were assessed prior to and on the last day of treatment for changes in serum creatinine, albumin, total protein, and urine-specific gravity. Horses underwent endoscopic examination of the esophagus, stomach, and pylorus prior to and 24 hours after the last treatment. A significant change in serum creatinine and total protein was observed on day 10 of treatment. No other significant findings were noted during the experiment. Results indicated that co-administration of phenylbutazone and firocoxib may cause renal disease.
PMID: 24354928

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Equinosis Gait Evaluation System

Rogue Equine Hospital is pleased to introduce the Equinosis® Gait Evaluation System.  It is the culmination of almost 20 years of research on gait analysis at the University of Missouri’s Colleges of Veterinary Medicine and Engineering with the support of the E. Paige Laurie Endowed Program in Equine Lameness. The system objectively detects and quantifies body movement asymmetry in a horse using small, wireless, body-mounted inertial sensors and a hand-held tablet PC. Instrumentation of the horse is quick, easy, and completely non-invasive. Data collection is in real time and veterinarians are free to perform their usual lameness evaluation routine without distraction.

Visit www.equinosis.com,
see more at: http://equinosis.com/#sthash.uUX3I3Y1.dpuf or call the office @ 541.826.9001.

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Manuka Honey and Wound Care

Manuka Honey and Wound Care
by Nancy Loving

With increasing emphasis by horse owners on approaching their horses’ health issues through more “holistic” and “natural” strategies, one such “natural” and relatively inexpensive treatment might include the use of honey for wound care. As a veterinarian you need to understand the physical and financial aspects of potentially using this option.

Honey application to cutaneous wounds is far from a “new” treatment; honey has been used since Egyptian times dating as far back as 2,000 B.C. as a means of managing wounds and inhibiting bacterial infection.

Yet it is important to know that not all honey is created equal. Manuka honey, derived from floral sources Leptosperum spp in New Zealand and Australia, has specific antibacterial and antioxidant properties that are absent in other honeys. Manuka honey is reported to have osmotic and pH effects; for example, it creates a more acidic pH environment that counteracts the alkaline pH of an infected wound, which is helpful for wound contraction. By lowering wound pH, protease activity is decreased and fibroblast activity and oxygen release are increased, all of which facilitate wound healing.

In addition, while bacterial-generated biofilm is known to impair healing, manuka honey has potent anti-biofilm properties: methylglyoxal, the bactericidal component of manuka honey, kills biofilm-embedded bacteria.

With the resurgence of the use of honey for wound care, licensed, medical-grade manuka honey is commercially available in therapeutic wound dressings: Medihoney  (Derma Sciences) and Active Manuka Honey UMF 18+ (Manuka Honey USA). A medical-grade product is one that has been “sterilized by gamma irradiation and has a standardized antibacterial activity.”

Use of non-sterilized honey has the potential to contaminate a wound with aerobic bacteria or fungi, therefore it should not be used.

Application of medical-grade manuka honey on a wound has the potential to reduce both the duration and expense of systemic antibiotic treatment while achieving favorable therapeutic results for the patient and client.

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“Nutritional Management of Insulin Resistance in Horses”

Nutritional Management of Insulin Resistance in Horses

By Alexandra Beckstett, The Horse

Managing Editor December 9, 2013 8:00

When it comes to caring for insulin-resistant (IR) horses, diet plays a very important role in managing insulin levels and preventing associated diseases such laminitis. Insulin resistance and hyperinsulinemia are key features of equine metabolic syndrome (EMS) and can also occur in horses with pituitary pars intermedia dysfunction (or equine Cushing’s disease). These animals often need to lose weight and consume fewer carbs.

To that end, Ray Geor, BVSc, PhD, Dipl. ACVIM, a professor and researcher at Michigan State University’s department of large animal clinical sciences, shared best feeding practices for IR horses at the 2013 International Equine Conference on Laminitis and Diseases of the Foot, held Nov. 1-3 in West Palm Beach, Fla. He first discussed weight loss, then carbohydrate consumption.

Managing Obesity

Geor explained that dietary restriction and exercise are two key components to curbing obesity in IR horses. “Dietary restriction is not rocket science,” he said. “But it requires owner/trainer compliance and patience and sticking to the program (to be successful). Diet changes are likely a lifelong pursuit, especially for horses with EMS that are easy keepers and have a tendency to become obese.”

Geor recommended owners of affected horses institute a weight loss program with the goal of improving the animal’s metabolism to reduce his risk of developing associated laminitis. He offered the following guidelines for developing a program:

Base your horse’s diet on forage or a forage substitute, and eliminate grain and calorie-dense feeds (e.g., sweet feeds) if possible. – Feed a lower-quality, low-energy forage, such as late-maturity hay, at between 60 and 80% of your horse’s daily energy requirements based on his body weight.
When feeding a low-quality forage, add a ration balancer to help your horse meet his vitamin E, copper, zinc, and other requirements. “Alternatively, forage-based, low-calorie feeds that contain added vitamins and minerals are now available commercially,” Geor said. “This type of feed offers convenience and may be used as a substitute for hay or fed as a component of the ration along with hay.”
Although all weight loss programs need to be individually tailored, as a general guide start by restricting your horse’s daily dry matter intake (DMI) to 1.5% of his body weight in total feed. The rate of weight loss will vary between horses but in general at least six to eight weeks of dietary restriction is needed for noticeable weight loss to occur, Geor said. If your horse’s weight loss response is less than desired, you might need to restrict his DMI to 1.25% body weight. If he still doesn’t lose weight after another six to eight weeks, you can restrict his DMI even further to 1% body weight, but Geor recommended never feeding lower amounts than that for health and behavior reasons. “Hay feeders and slow feeders (e.g., hay nets with multiple small holes) are one way to extend your horse’s hay supply if you’re not feeding much,” he said.
Restrict or eliminate pasture grazing using a grazing muzzle or a drylot for turnout. “It should be noted that simply restricting the time allowed for grazing may not be an effective strategy for weight loss,” Geor cautioned. “Ponies have been observed to consume up to 1% of body weight within three hours of pasture turnout.”
And as with any diet changes, make feed changes gradually and avoiding withholding food from your horse for long periods. Divide rations into three to four small meals, and do not bed a dieting horse on straw or shavings to reduce the risk of him eating them and potentially suffering an impaction, Geor said.
Geor encouraged owners to use a number of simple tools to monitor their horses’ weight loss, including body condition scoring and measurements of girth and abdominal (‘belly’) circumference. “Body condition score is not always a sensitive indicator of weight loss,” he added. “However, girth and belly circumference measurements will decrease with weight loss, and I recommend recording these measurements at three- to four-week intervals.”

But once your horse meets his targeted weight and condition, you can’t just stop there: “Develop and continually update an appropriate weight maintenance program,” Geor said. “It should include monthly assessment of body weight and BCS to ensure that the feeding program is appropriate for the current level of physical activity and other environmental influences on energy requirements.”

Controlling Carbohydrates

Veterinarians and researchers have long associated laminitis with increased nonstructural carbohydrate (NSC) intake—especially in pastured horses. And because NSCs can contribute to exaggerated insulin responses, it’s particularly important to restrict their intake in IR horses.

Geor offered suggestions for controlling NSC intake:

Feed a forage-based diet with a low NSC content (less than 12%). “Soaking hay can help lower NSC levels, but it’s not a panacea,” he said.
Eliminate grain and sweet feed (both high in NSCs) from the diet.
Restrict or eliminate pasture access. At certain times of year (e.g., during spring and early summer growth periods, after summer or fall rains, or after drought or frost) pasture forages’ NSC content is quite high, so Geor advised keeping IR horses off pasture during these periods to reduce the risk of developing laminitis. He said a grazing muzzle might be the best way to safely restrict grass consumption (studies show it can decrease pasture intake by about 80%), as simply providing a smaller space to graze can lead to an overgrazed area, which will still have high NSC content due to stress. Also, “beware the ‘Hoover’ pony,” Geor said. “They can adapt and consume around 40% of their daily DMI in just two to three hours.”
In conclusion, Geor said correcting obesity and restricting NSCs are not a cure for insulin resistance but these strategies can help to prevent laminitis in affected horses.

Disclaimer: Seek the advice of a qualified veterinarian before proceeding with any diagnosis, treatment, or therapy.

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Diagnoses, clinical pathology findings, and treatment outcome of geriatric horses: 345 cases (2006-2010).

Diagnoses, clinical pathology findings, and treatment outcome of geriatric horses: 345 cases (2006-2010).

J Am Vet Med Assoc. 2013 Dec 15;243(12):1762-8

Authors: Silva AG, Furr MO

Abstract
Objective-To compare clinical, clinical pathology, and outcome variables between geriatric and nongeriatric horses. Design-Retrospective case-control study. Animals-690 horses (345 horses ≥ 20 years old and 345 horses > 1 and < 20 years old) examined at a referral hospital. Procedures-Medical records were examined, and data collected included horse description, diagnosis, outcome, and CBC and serum biochemical analysis results. Cases were horses ≥ 20 years old, and controls were horses > 1 and < 20 years old. Results-Mean ± SD age was 23.9 ± 4.6 years for cases and 9.2 ± 3.6 years for controls. Arabian and pony breeds were significantly overrepresented in the geriatric group, compared with the control group. Diagnoses related to the digestive system, musculoskeletal system, and respiratory system were most common in this hospital population overall (cases and controls). Colic was the most common health problem overall. Digestive system disorders were significantly more prevalent among cases. Short-term survival rates for most categories of colic were no different for cases than for controls, with the exception of the category idiopathic colic. Considering all conditions, cases were significantly more likely to be nonsurvivors than were controls. Minor differences in serum biochemical results were found in some disease subcategories. Geriatric horses with colic were not more commonly euthanized than were adult nongeriatric horses. Conclusions and Clinical Relevance-Results indicated that in this population of horses in a referral hospital, age was associated with the prevalence of specific disease conditions. Few differences between cases and controls were found in serum biochemical values. PMID: 24299549 [PubMed - in process]

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Pain Management for Horses with Laminitis: Where Are We Now?

Pain Management for Horses with Laminitis: Where Are We Now?
By Alexandra Beckstett, The Horse Managing Editor
Updated: Monday, December 2, 2013
 
Originally published on TheHorse.com

When an owner makes the difficult decision to euthanize a horse with laminitis, it’s often because the horse is simply in too much pain to justify prolonging treatment. For this reason, researchers are continually trying to come up with improved analgesic (pain relief) methods. Andrew van Eps, BVSc, PhD, MACVSc, Dipl. ACVIM, senior lecturer in Equine Medicine at The University of Queensland School of Veterinary Science, reviewed current pain management options for laminitic horses at the 2013 International Equine Conference on Laminitis and Diseases of the Foot, held Nov. 1-3 in West Palm Beach, Fla.

The pain laminitic horses experience might stem from several sources, van Eps said, including inflammation, submural (beneath the hoof wall) pressure, tearing of submural tissues, ischemia (lack of blood flow), the distal phalanx (P3, or coffin bone) applying pressure to the sole, and neuropathic mechanisms (those caused by nerve damage and described, in humans, as throbbing, stabbing, sharp pain).

We, as humans, want to control horses’ pain not only for welfare reasons but also because it interferes with the animal’s function. However, we can’t simply eliminate pain completely, as it does serve an important purpose in self-preservation, van Eps said.

“Without pain, weight-bearing and ambulation are not restricted in horses with laminitis, so consequently there can be increased mechanical distractive forces on the lamellar tissue and progression of the lesion itself,” he explained.

The first step in determining how to manage a laminitic horse’s discomfort is to assess his pain level objectively and routinely, said van Eps. This might include:

Performing a lameness exam.
Observing the horse’s posture and stance.
Hoof testing, although this can vary among cases, van Eps cautioned.
Obtaining systemic measurements such as heart rate, respiratory rate, and blood pressure. “These are reasonably sensitive, especially in hospital settings where horses are monitored serially,” he said.
Using tools such as force plates and pedometers to evaluate weight distribution. “Incessant shifting of weight is one of the first and most subtle signs of laminitis pain,” van Eps said. “Recently, the use of human pedometers (to document weight-shifting frequency) … proved to be much more sensitive than visual assessment alone.”
Performing behavioral analysis. “Serial recording of events such as teeth grinding, interactions with people, and position in the stall (back corner vs. close to the door) can be useful markers of pain that are more subtle,” he said.
Van Eps said owners and veterinarians also need to consider disease stage and what they are trying to achieve—long-term or short-term pain relief. Then they can choose from the two categories of analgesic therapies what makes the most sense for that individual horse: a systemic or regional approach.

Systemic Therapy
Systemic therapies are those that travel through the bloodstream. This primarily involves non-steroidal anti-inflammatories (NSAIDs) such as phenylbutazone, flunixin meglumine, and ketoprofen. While this drug class is potent and effective in most acute and chronic laminitis cases, van Eps said it’s also associated with side effects such as gastric/colonic ulcers and kidney damage because it targets not only the pain and inflammation-causing COX 2 enzyme, but also the protective COX 1 enzyme.

Thus, “more COX 2-selective NSAIDs, such as firocoxib and meloxicam, are potentially associated with fewer side effects,” he said. “However, their analgesic efficacy and long-term safety compared with non-COX-specific NSAIDs requires further investigation.” He added that some studies have recently confirmed these drugs are both safe and effective, although expensive.

When NSAIDs alone are insufficient, van Eps suggested using a combination of other systemic drugs to provide additional analgesia.

“The concept of multimodal analgesia involves the use of different types of analgesic drugs that act by different mechanisms at different sites in the nervous system to provide superior analgesia with fewer side effects,” he explained. “The use of multimodal analgesia is often reserved for laminitis cases that do not respond to conventional therapy.”

Drugs van Eps said veterinarians might use in a multimodal approach including:

Intravenous (IV) lidocaine, which he said can be effective for acute laminitis, but comes with a risk of toxicity/neurologic sign development during infusion if an overdose occurs. “This can happen if the horse receives even a transient overdose (inadvertent increase in the infusion rate),” van Eps explained. “Therefore this therapy is best done in hospital situation with infusion pumps and a dedicated IV catheter.”
Opioids, which van Eps said are not particularly effective alone, but might be useful in combination with drugs such as lidocaine, ketamine, morphine, detomidine, and acepromazine;
Ketamine, an anesthetic that acts through decreasing central sensitization;
Alpha-2 agonists, which he described as short-lasting sedatives useful for managing unrelenting pain; 
Amitriptyline, which van Eps said has been used effectively as an antidepressant and analgesic in humans, but not in horses; and
A promising anti-inflammatory (soluble epoxide hydrolase inhibitor) currently under investigation at the University of California, Davis.
The last systemic therapy van Eps described was gabapentin, a drug originally used to treat neuropathic pain and seizures in humans. Much is still unknown about this drug’s effects and appropriate dosage in horses, although he said IV or oral gabapentin might be useful when treating acute laminitis cases.

“We routinely administer gabapentin to clinical cases with acute and chronic laminitis without ill-effect,” he said. “Unfortunately, it is very difficult to accurately assess whether gabapentin improves analgesia in these cases.

“The main side effect of gabapentin administration in horses is mild sedation and tranquilization,” he added. “However, this may actually be of benefit in acute laminitis cases, as recumbency and reduced ambulation are encouraged, as they protect the tissues from further damage caused by weight-bearing.”

Regional Therapy
Regional analgesic techniques, as the name implies, aim to treat a specific area. Examples of regional methods used in laminitis cases include:

Continuous peripheral nerve block of the palmar nerves. Van Eps said veterinarians might use this method when performing a painful procedure such as hoof resection.
Epidural analgesia, in which the veterinarian administers long-term pain management (e.g., opioids, alpha-2 agonists, local anesthetics) via an epidural catheter. “It can be useful to control pain associated with hind-limb laminitis,” van Eps said. “Morphine is probably the most useful, as its analgesic effects are long-lasting (12-14 hours) and it does not cause ataxia (incoordination).”
Therapeutic hypothermia, or cryotherapy, has both anti-inflammatory and nerve conduction effects.
Take-Home Message
Because there’s no one-size-fits-all formula to managing laminitic horses’ pain, van Eps recommended owners work with their veterinarians to tailor a multimodal approach for each individual case. “Equine analgesia is evolving,” he said. “There’s more evidence for COX 2 selective drugs than 10 years ago, and now that we recognize the contribution of neuropathic pain in laminitis, we’re beginning to investigate drugs that can help to control this.”

Disclaimer: Seek the advice of a qualified veterinarian before proceeding with any diagnosis, treatment, or therapy.

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Snake Bite in Horses

From Equine Disease Quarterly, Univ. Of KY.

Snakebite in Horses

With warm weather comes the increased risk of snakebite. The major venomous snakes in the United States are the pit vipers, including rattlesnakes, water moccasins, and copperheads. Pit vipers are named after the heat-detecting holes, or pits, on each side of the head that help the snake locate prey. Pit vipers can be differentiated from other snakes by their triangle-shaped heads, narrowed necks, and tail rattles (rattlesnakes only). Coral snakes, another type of poisonous snake in the U.S., do not pose much risk to horses because of their small mouth size.Venom components vary tremendously by snake species, but most venoms contain substances that cause breakdown of tissues and blood vessels, impair blood clotting, and damage the heart. Venoms from some species of snake also contain neurotoxins. Snakebite severity depends on multiple factors such as snake species, size, recent feeding, and number of bites. Some bites are “dry bites,” where little venom is injected. Other bites, such as when a snake is stepped on and releases all of its venom agonally, can be very severe. Victim factors such as horse size, age, disease conditions, medications, and bite location also influence bite severity.Clinical signs of snakebite in horses vary widely but generally include pain and swelling at the bite site, and often sloughing of tissues near the bite. Bite wounds may not be readily apparent. Dry bites with little venom injected or bites from copperhead snakes often cause only mild signs. Bites from dangerous species of snakes and large doses of venom can cause marked pain and swelling, coagulopathy, hemorrhage, cardiac arrhythmias, shock, collapse, and even death. With neurotoxic venoms, paralysis can occur. Horses bitten on the nose can develop nasal swelling and respiratory distress. Signs of envenomation can occur within minutes or be delayed for many hours.The best first aid is to keep the horse calm and arrange for immediate veterinary care. No first-aid treatments performed by owners in the field have proven particularly helpful, and many folk remedies can even be harmful. Suction devices have not been shown to be beneficial in animal models of snakebite.Treatment varies with the severity of the bite, but may include fluids, pain medications, wound care, antibiotics, tetanus prophylaxis, and antivenin. Antivenin can decrease the amount of tissue damage and hasten recovery times, and can be especially helpful in cases of severe envenomation. Antivenin is dosed according to the estimated amount of venom injected, not the patient size, so even one vial of antivenin can have beneficial effects. Cardiac arrhythmias occur in many horses and may require treatment. Horses with severe nasal passage swelling may need treatment to maintain a patent airway; nutritional support may be required if swelling impairs the horse’s ability to eat and drink.Even after horses have recovered from the immediate effects of snakebite, subsequent complications such as heart failure or kidney damage are possible. Cardiac failure can occur weeks to months after the bite incident, necessitating continued evaluation and monitoring.A vaccine is now available for use in horses to help prevent complications of snakebite, but efficacy in horses is not yet well documented. Contact your veterinarian for more information about snakebite in your region.

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“Differential Virulence and Pathogenesis of West Nile Viruses.”

Pathogenesis of West Nile Viruses.
pubmed: pubmed horse / by Donadieu E, Bahuon C, Lowenski S, Zientara S, Coulpier M, Lecollinet S / 18 hours ago
Differential Virulence and Pathogenesis of West Nile Viruses.

Viruses. 2013;5(11):2856-2880

Authors: Donadieu E, Bahuon C, Lowenski S, Zientara S, Coulpier M, Lecollinet S

Abstract
West Nile virus (WNV) is a neurotropic flavivirus that cycles between mosquitoes and birds but that can also infect humans, horses, and other vertebrate animals. In most humans, WNV infection remains subclinical. However, 20%-40% of those infected may develop WNV disease, with symptoms ranging from fever to meningoencephalitis. A large variety of WNV strains have been described worldwide. Based on their genetic differences, they have been classified into eight lineages; the pathogenic strains belong to lineages 1 and 2. Ten years ago, Beasley et al. (2002) found that dramatic differences exist in the virulence and neuroinvasion properties of lineage 1 and lineage 2 WNV strains. Further insights on how WNV interacts with its hosts have recently been gained; the virus acts either at the periphery or on the central nervous system (CNS), and these observed differences could help explain the differential virulence and neurovirulence of WNV strains. This review aims to summarize the current state of knowledge on factors that trigger WNV dissemination and CNS invasion as well as on the inflammatory response and CNS damage induced by WNV. Moreover, we will discuss how WNV strains differentially interact with the innate immune system and CNS cells, thus influencing WNV pathogenesis.
PMID: 24284878 [PubMed – as supplied by publisher]

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